Mihai Miclăuș



Timeline

Team Lead – Balmus Lab Romania, Transylvanian Institute of Neuroscience, Cluj-Napoca, Romania

Bioinformatician – QIAGEN, Cluj-Napoca, Romania

Principal Investigator – National Institute of Research and Development for Biological Sciences, Cluj-Napoca, Romania

Postdoc – Rémy Bruggmann Lab, University of Bern, Switzerland

PhD – Joachim Messing Lab, Rutgers University, USA


Mihai Miclăuș

Mihai has started his research career in the wet-lab, having a PhD in molecular biology, and started shifting to dry-lab in 2015. He spent 10 years in academia as principal investigator in the field of genomics and genetics, followed by two years at QIAGEN, as bioinformatician.

He joined the Balmus Lab in the summer of 2023 as senior bioinformatics scientist, leading the Romanian hub of the lab. He is interested in deciphering the molecular complexities underlying Huntington's disease. This endeavor entails bioinformatics work, encompassing transcriptomics, proteomics, GWAS, and other interconnected 'omic' analyses. 


Selected publications

Maize cytolines as models to study the impact of different cytoplasms on gene expression under heat stress conditions. full text

Ardelean, I.V., Bălăcescu, L., Sicora, O., Bălăcescu O., Mladin, I., Haș, V., Miclăuș, M. (2023), BMC Plant Biol 23, 4

Maize cytolines unmask key nuclear genes that are under the control of retrograde signaling pathways in plants. full text

Miclaus, M., Balacescu, O., Has, I., Balacescu, L., Has, V., Suteu, D., Neuenschwander, S., Keller, I., Bruggmann, R. (2016), Genome Biology and Evolution 8 (11) pp. evw245-15

Romanian maize (Zea mays) inbred lines as a source of genetic diversity in SE Europe, and their potential in future breeding efforts. full text

Șuteu, D., Bacila, I., Has, V., Has, I., Miclăuș, M. (2013), PLoS One 8(12): e85501

Differential gene expression and epiregulation of alpha zein gene copies in maize haplotypes. full text

Miclăuș, M., Xu, J-H., Messing, J. (2011), PLoS Genetics 7(6): e1002131

The maize high-lysine mutant opaque7 is defective in an acyl-CoA synthetase-like protein. full text

Miclăuș, M., Wu, Y., Xu, J-H., Dooner, H.K., Messing, J. (2011), Genetics 189: 1271-1280